Is atransparentlight-transforming laminated
structure comprising photoreceptors,
interneurons and ganglion cells overlying the
retinal pigment epithelium.
Superficial retinal vessels form four major
arcades over the surface of retina.
Within the suprachoroidal space are the long
ciliary nerves and arteries which can be seen
peripherallyat 3 and 9 o’ clock position
Retina
3.
Normal attachmentsare strongest at the
◦ Disc, the fovea and especially the ora serrata/vitreous
base which remains adherent even when posterior
vitreous detachment is otherwise complete.
Abnormal attachments
◦ Area of lattice degeneration, white without pressure,
pigment clumps and condensation around vessels
Vitreoretinal Adhesions
4.
RD isa relatively common sight-threatening
condition with an incidence of around
1/10,000/year.
Neurosensory retina is separated from retinal
pjgment epithelium is termed as RD.
Retinal Detachment
5.
Rhegmatogenous (Rhegma-break)
◦ Occurs secondarily to a full thickness defect in the sensory
retina which allow fluid derived from synchytic vitreous to gain
access to the subretinal space.
Tractional
◦ In which the NSRis pulled away from the RPE by contracting
vitreoretinal membranes in the absence of a retinal break
Exudative
◦ Is caused neither by break nor traction; the SRFis derived
fromfluid in the vessels of the NSR or choroid or both
Combined tractional-rhegmatogenous
◦ Is the result of a combination of a retinal break and retinal
traction
Types of RD
6.
It isa force exerted on the retina by structures
originating in the vitreous, and may be dynamic or
static
Dynamic
◦ Is induced by eye movements and exerts a cetripetal
force towards the vitreous cavity. It plays an important
role in the pathogenesis of retinal tear and
rhegmatogenous RD
Static
◦ Is independent of ocular movements. It play a key role in
the pathogenesis of tractional RD and proliferative
vitreoretinopathy
Vitreoretinal Traction
7.
PVD isa separation of the cortical vitreous from the
internal limiting membrane.
It can be classified as
◦ Onset
Acute PVD is by far the most common. It develops suddenly and
usually becomes complete soon after onset. Chronic PVD occurs
gradually and may take weeks or months to become complete
◦ Extent
Complete
PVD in which the entire vitreous cortex detaches up to the posterior
margin of the vitreous base
Incomplete
PVD in which residual vitreoretinal attachements remain posterior to
the vitreous base
Posterior Vitreous
Detachment
8.
Is afull thickness defect in the sensory retina
It can be classified according to
◦ Pathogenesis
◦ Morphology
◦ Location
Retinal Break
9.
Tears
◦ Causedby dynamic vitreoretinal traction and have a
predilection for the superior fundus
Holes
◦ Caused by chronic atrophy of the sensory retina and
may be round or oval. They have predilection for the
temporal fundus
Pathogenesis
10.
U-Tears
◦ (Horseshoe,flap or arrowhead) consist of a flap, the apex of which is
pulled anteriorly by the vitreous, the base remaining attached to the
retina
Incomplete U-Tears
◦ Which may be linear, L-shaped or J-shaped are often paravascular
Operculated Tears
◦ In which the flap is completely torn away from the retina by detached
vitreous gel
Dialyses
◦ Are circumferential tear along the ora serrata with vitreous gel
attached to the posterior margins
Giant Tears
◦ Involve 90 or more of the circumference of the globe
Morphology
11.
Oral
◦ Theyare located with in the vitreous base
Post-oral
◦ They are located between the posterior border of the
vitreous base and equator
Equatorial
◦ At or near the equator
Post-equatorial
◦ Behind the equator
Macular
◦ Are at the fovea
Location
14.
Is usuallyan ophthalmic emergency
Untreated it usually progresses to blindness
even pthysis
With appropriate treatment it may have an
excellent outcome
Is the commonest form of RD
It occurs when break is allow to liquefaction
vitreous to enter with in sub retinal space and
lift the NSR from RPE
Rhegmatogenous RD
15.
Flashes (usuallytemporal and more noticeable in dim
conditions)
Floaters
Curtain like field defect
Dec. VA
Retinal break upper temporal quadrant is the
commonest location
RD unilateral corrugated convex dome shaped of retina
and loss of RPE/choroidal clarity
Chronic signs, retinal thinning, demarcation line from
3months, intra retinal cyst from 1 year may also have
RAPD if extensive RD, relative field defect, dec. IOP
Clinical Features
16.
Work up Toreach Diagnosis
Visual
Symptoms
Asymptomatic, flashes, floaters, curtain field defect,
decreased VA
POH Refractive error, Surgery, Laser treatment, trauma
PMH Connective tissue syndromes, diabetes,
FH Retinal problems/detachment, connective tissue syndrome
SH Driver, other occupation
VA Best-corrected/pin-hole
Cornea Clarity (for surgery)
AC Cells/flare
Pupil RAPD for extensive detachment
Lens Cataract
Tonometry IOP may be low, normal or high
Vitreous Hemorrhage, pigment (tobacco dusting)
Fundus RD; Location, extent, age (atrophy, intraretinal cyst, pigment
demarcation line, PVR, Retinal break, associated degeneration
17.
Consider USif unable to adequately
visualize(dense cataract, hemorrhage)
B-Scan US Highly reflective irregular convex
membrane
Work-up to reach diagnosis
18.
Ocular General Trauma
(blunt/penetrating,
Surgery
RefractiveMyopia
Lenticular Aphakia,
Pseudophakia,
posterior capsulotomy
Retinal Lattice degeneration,
Retinoschisis, Retinal
necrosis
Other eye H/O RD especially Giant
retinal tear
Systemic Stickler’s syndrome,
Marfan’s syndrome
Ehlers-Danlos
Risk Factors for RRD according to other
ocular and systemic features
19.
Retinal thinning
Demarcation lines (high water marks)
Intra retinal cysts
Proliferative vitreoretinopathy
Features of Chronic RRD
20.
The mainaim of surgery is to successfully
repair the detachment with minimal trauma
and attendant risk
To seal the hole and close the retinal breaks
Principles of Retinal Surgery
21.
Is asurgical procedure in which material
sutured on to the sclera (explant) creates an
inward indentation(Buckle).
Its purposes are to close retinal breaks by
apposing the RPE the sensory retina and
reduce dynamic vitreoretinal traction at site s of
local vitreoretinal adhesions
Principles of Scleral Buckkling