This presentation provides an overview of syncope, a common medical emergency in dental practice. Created during my internship, this presentation aims to educate dental students on the causes, symptoms, diagnosis and management of syncope with a focus on dental specific considerations.
SS Y NC O P E
Syncope
Kingshika Joylin
CRRI’24
For the dept. of oral and maxillofacial surgery
Rajas dental college and hospital, Tirunelveli dt.
in dentistry.
2.
Definition
Transient, self limitedloss of consciousness due to acute global impairment of cerebral blood flow.
(Harrison)
AKA ‘vasovagal syncope’ or ‘vasodepressor syncope’
Reflex/Neural mediated Orthostatichypotension Cardiac
• Most common in occurrence
• Due to episodic vasodilation
& loss of vasoconstrictor
tone
• Usually a prodrome
Transient change in cardiovascular
homeostatic reflexes
o VASOVAGAL SYNCOPE
- orthostatic vasovagal
- emotional vasovagal (fear,
phobia, pain)
o SITUATIONAL SYNCOPE
(defecation, swallowing,
micturition, coughing)
o CAROTID SINUS SYNDROME
e.g., tie too tight
Chronic impairment in
cardiovascular homeostatic reflexes
o VOLUME DEPLETION
haemorrhage, vomiting, diarrhoea
o AUTONOMIC FAILURE
- Primary autonomic failure
(old age, parkinsonism)
- Secondary autonomic failure
(diabetes, amyloidosis, spinal cord injuries)
- Drug induced
(vasodilators, diuretics, anti-depressants)
o ARRHYTHMIA
(bradyarrhythmia, tachyarrhythmia)
o STRUCTURAL DEFECT
(aortic stenosis, prosthetic valve
dysfunction, MI)
o GREAT VESSEL DEFECT
(pulmonary embolism, acute aortic
dissection)
Often feels dizzy when changing position
5.
Pathophysiology
Anxiety
Catecholamine release
Peripheral vascularresistance
Pooling of blood in periphery
Stimulation of sympathetic
nervous system
Heart rate
Feeling of warmth
Rapid breathing
Pallor
Perspiration
Decompensation
Reflex vagally mediated
Cerebral blood flow
SYNCOPE
Bradycardia
Weakness
Nausea
Hypotension
Arterial blood pressure
6.
Stimulus sent to
nucleiof vagus nerve
Response sent via
parasympathetic nervous
system to the heart
Cardio inhibitory
response: BP
Less O2 carried back to
brain resulting in
‘SYNCOPE’
7.
X Failure ofcerebral blood flow autoregulation leads to
global cerebral hypoperfusion
Cerebral blood flow normally, 50-60 ml/min/100gm of brain tissue
If reduced, 25 ml/min/100gm Impaired consciousness leading to complete if
prolonged for 6-8 seconds
8.
Sympathetic vs Parasympathetic
Decreasedvenous
return to the heart
Normally, this sympathetic
stimulation compensates
and patient does not faint
In other hand, Stress causes strong left ventricular
contraction that activates cardiac mechanoreceptors &
vagus nerve resulting in vasodilation, decreased heart rate ,
hypotension leading to syncope
Baroreceptors in the carotid sinus and arch of aorta
signal to the brain to produce more adrenaline to
increase BP &HR
9.
Sudden emotional stress
Pain,especially sudden & unexpected
Anxiety
Sight of blood or surgical or other dental instruments
Erect sitting or standing posture
Exhaustion
Hot, humid, crowded environment
Poor physical condition
Precipitating
factors
Fasting
Low circulatory volume
Poor physical conditions
Warm & crowded environment
Aggravating factors
10.
Prodromal symptoms
E AR L Y L A T E (established syncope)
Feeling of warmth
Loss of colour: pale or ashen grey skin
Diaphoresis
Reports of feeling bad
Nausea
Blood pressure at baseline level or
slightly lower
Tachycardia
Pupillary dilation
Yawning
Hyperpnea
Cold hands & feet
Hypotension
Bradycardia
Visual disturbances
Dizziness
Loss of consciousness
11.
San Francisco Syncoperule
‘C H E S S’
• Congestive heart failure history
• Hematocrit < 30%
• ECG (abnormal)
• Shortness of breath
• Systolic BP <90mmhg
Higher risk for such patients
Adverse outcomes include death, MI, arrhythmia, pulmonary embolism, stroke, subarachnoid
haemorrhage
12.
Management
Four stages ofsyncope management;
• Presyncope
• Syncope
• Delayed recovery
• Post syncope
13.
‘Presyncope’
STEP 1
Supine position
Movinglegs vigorously
STEP 2
Circulation -> Airway -> Breathing
Assessed as being adequate
STEP 3
(Definitive care)
O2 administered using full face mask
Spirit of ammonia
14.
‘SYNCOPE’
Step 1: POSITION
•Terminate the dental procedure
• Supine position with legs slightly elevated
• Muscle movements helps increase the return of blood from the periphery
‘Trendelenburg position’
15.
Step 2: C-A-B(circulation-airway-breathing)
To assess circulation,
‘Carotid pulse’ is palpated
In syncope,
• Weak, thready carotid pulse
• Slow heart rate
To assess airway,
• Adequate airway present when the patient’s
chest moves and exhaled air can be heard &
felt
To assess breathing,
• Spontaneous respiration is usually evident
• Rescue breathing may be necessary
‘HEAD TILT-CHIN LIFT’ METHOD
Positioning of the victim + Airway patency -> Rapid return of consciousness
‘LOOK, LISTEN, FEEL’
16.
StepStep 3: Definitivecare
• Administration of O2
• Monitoring of vital signs (BP, heart rate, respiratory rate)
• Additional procedures
17.
• Loosening ofbinding clothes
• Use of a respiratory stimulant, ‘AROMATIC AMMONIA’
• Cold towel placed on the forehead of a patient who is warm & blankets for those who feels cold
• If bradycardia, anticholinergic such as Atropine i.v or i.m
• Stimulus that precipitated the episode (syringe, an instrument or a piece of bloody gauze)
• Frequently, hypoglycemia is involved - Administration of ‘sugar’ in form of orange juice or non diet soft drink
18.
‘Delayed recovery’
• Ifthe patient does not recover in 15-20 mins call for emergency medical services (EMS)
• Perform Basic life support (BLS)
• CAUSES
- Seizure
- Cerebrovascular accident (stroke)
- Transient ischemic attacks (TIA)
- Cardiac dysrhythmias
- Hypoglycaemia
19.
‘Post syncope’
• Notany additional dental treatment
• Body may take 24 hr to settle down
• Should determine the precipitating factors
• Arrangements for adult escort to take patient home
20.
References
• Little andFalace’s Dental management of the medically compromised patient - 9th
edition
• SM Balaji’s Textbook of oral & maxillofacial surgery - 3rd
edition
• Stanley F Malamed’s Medical emergencies in the dental office
• Harrison’s manual of medicine - 19th
edition